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doi: 10.3390/biomedicines9070792

The vascular endothelium, a critical regulator of vascular tone, becomes dysfunctional when excessive ROS impairs nitric oxide (NO) bioavailability, a hallmark of HTN

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Paxillin, located at the interface between the plasma membrane and the actin cytoskeleton, is controlled by miR-137, miR-145, miR-218 and miR-125b [88, 89]

Sulfation (SULT Enzymes) Sulfotransferases (SULTs) transfer a sulfonate group (SO3) from the cofactor 3'-phosphoadenosine-5'-phosphosulfate (PAPS) to the hydroxyl or amine group of the substrate