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While additional research is necessary to explore the potential role of GLP-1Rs expressed outside of the beta cell, including within the enteric and peripheral nervous systems, in enhancing beta cell function through enteral glucose mediation, current evidence indicates that the central nervous system (CNS) GLP-1Rs are not essential for the physiological regulation of blood sugar levels by endogenous GLP-1 or the pharmacological control of glucose homeostasis dependent on GLP-1Rs
The study showed a clear dose-response: 5 sessions produced 26.5% regrowth versus 10% with 3 sessions (p = 0.0025)
The treated brains still lost volume, so this slowed the loss rather than restoring tissue, and the benefit was tied to having high homocysteine to begin with
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