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It was suggested that polymorphisms of glutathione transferases (Ile105Val polymorphism of the GSTP1 gene), ATP binding cassette transporters, OCT2, cytochrome P450 enzymes, polymorphism of ABCC2 (ATP binding cassette subfamily C member 2), and AGXT (alanine glyoxylate aminotransferase) may be involved in the development of OIPN [44,45,46,47,48,49,50]
Enzymes involved in GSH biosynthesis and function like -glutamyl-cysteine ligase and glutathione synthetase are completely dependent on ATP and require magnesium as a cofactor (Bani Younes et al., 2020
Toxicol Sci 2005;83:1906 [DOI] [PubMed] [Google Scholar] 76.DeLeve LD, Wang X, Kaplowitz N, Shulman HM, Bart JA, van der Hoek A.Sinusoidal endothelial cells as a target for acetaminophen toxicity
K.MantriS.et al (2010)
It was also discovered that these peptides may effectively destroy BC cells in vivo as well as in vitro
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