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Doxorubicin induces senescence or apoptosis in rat neonatal cardiomyocytes by regulating the expression levels of the telomere binding factors 1 and 2

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GHK-Cu and tissue repair-associated signaling An experimental investigation [8] evaluated the implications of the GHK-Cu peptide complex on tissue repair-related processes using preclinical wound models

After surgical perturbations in enterocytic integrity, elevated cellular growth and differentiation, which occur within days, is followed by temporal organ-specific adaptations to the bypass (148)