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A population- based study from Olmsted County, Minnesota, published in 2011, showed that patients with specialized intestinal metaplasia of the EGJ have a significantly lower risk for EAC compared with patients who have BE

We hypothesize this to be related to the peptide increasing progesterone as a consequence of elevated growth hormone (GH) levels

Recent studies have revealed that dysregulation of mitochondrial dynamicsincluding impaired mitophagy, imbalances in mitochondrial fusion and fission, and defects in mitochondrial biogenesisis closely associated with CRC initiation, progression, and resistance to therapy [46]

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