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GHK-Cu provides essential support through optimization of collagen quality via TGF- pathway activation and copper delivery for lysyl oxidase-mediated cross-linking that determines tissue mechanical properties

It co-localizes with multiple hormones and neurotransmitters, suggesting broad regulatory functions

This gene modulation effect extends to pathways related to wound response, growth factors, and anti-fibrotic mechanisms, making it of growing interest in regenerative biology studies

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While the ipsilateral precursory lesion could be explained by an early focal invasion by immune cells that remain dormant for years, the contralateral mirror lesions are harder to ascribe to such a mechanism