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PE-22-28 is still in the early stages of investigation, but its ability to influence the nervous system at such a targeted level makes it one of the more intriguing compounds in the peptide conversation today

The dependency between the number of missing values for each protein with respect to protein intensities was also illustrated in Supplementary Fig

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The consequence of this interaction is the breakdown of immune tolerance, manifested by the proliferation of follicular helper T (Tfh) and Th17 cells, impaired function of Tregs, aberrant B-cell activation leading to excessive autoantibody production, elevated levels of inflammatory mediators, multiorgan inflammation, and tissue damage, ultimately culminating in the development of autoimmune disorders

In principle, GST-mediated drug resistance would be reversed by the addition of a reasonably nontoxic modulator