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BPC-157 affects the whole body through systemic activity across the gastrointestinal tract, central nervous system, vascular system, and musculoskeletal tissue, extending its effects well beyond the local injury site

Key ways it appears to mimic or amplify natural gastrointestinal and tissue repair mechanisms observed in animal studies: Strongly upregulates angiogenesis through multiple pathways (VEGF, EGR-1, FAK-paxillin) in injured tissues, similar to the bodys own response during normal wound healing and tendon/ligament repair

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In contrast, DKK1 expression was significantly decreased by proflavine to approximately 3.7-fold, the lowest observed (Fig