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As Lushchak [60] discussed, inhibition of glutathione reductase, phosphofructokinase, fatty acid synthase, or activation of fructose-1,6-bisphosphatase by CoASSG was shown

However, issues such as drug resistance and associated side effects have significantly restricted the clinical application of these treatments

This was paralleled by the accumulation of glucose-6-phosphate (6P) in steady state GSH-deficient FoB (Supplementary Fig

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However, the concentration of both forms of ascorbate was significantly lower compared with the control (AsA/DHA = 6.5) (Fig