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Attractive targets include members of the LOX family, namely LOX and LOX-like 2 (LOXL2), which mediate collagen and/or elastin crosslinking [97, 98], thereby contributing to ECM remodeling in the tumor microenvironment

Measurement of the NF-B activation in our in vivo experiment showed that GHK-Cu blocked the transcriptional activity of NF-B through suppression of NF-B by altering the phosphorylation of its inhibitor i-B

* It was reported to be a form of programmed cell death by the Nomenclature Committee on Cell Death (NCCD) in 2018

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