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The adoption of a pro-inflammatory phenotype in microglia can be driven by various factors, including the presence of pro-inflammatory mediators, infiltration of peripheral immune cells, pathological deposits (such as tau aggregates), and viral proteins [e.g., proteins trans-activator of transcription (Tat)] [26, 27]

They will also monitor your symptoms to see how they respond

Schewe, J

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Blocking these proteins results in caspase-8 activation and increased TNF sensitivity, thereby enhancing and accelerating apoptosis (Choi et al., 2022)