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This FOXO4-mediated nuclear retention of p53 is a key mechanism by which senescent cells maintain apoptosis resistance despite their dysfunctional state

The mechanisms are fundamentally different, and that difference has significant clinical implications

Loosey-goosey tissues (as I sometimes describe it to patients) need something to help them reorganize

He pulled together findings from journals such as Gastrointestinal Endoscopy, Current Pharmaceutical Design, Expert Opinion on Biological Therapy, and Wound Repair and Regeneration