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In a transgenic mouse model, depletion of FAP-expressing cells led to rapid hypoxic necrosis mediated by interferon-gamma (IFN-) and tumour necrosis factor-alpha (TNF-), both associated with CD8 + T cell-dependent tumour cell killing [47]

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To encapsulate, mitophagy is an evolutionarily conserved mechanism that identifies and degrades dysfunctional mitochondria in a timely manner, thereby mitigating oxidative stress and enhancing cellular energy

In certain conditions, this effect is considered beneficial as it can impede tumor growth, delay disease progression, and even lead to the elimination of some cancer cells through the induction of an immune response [55]

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