Cagrilintide is a long-acting acylated agonist of amylin receptor [1]
The full ingredient list: Ingredients: Aqua (Solvent), Tetrahexyldecyl Ascorbate (Antioxidant), Dimethicone (Skin conditioning agent), Glycerin (Humectant), C9-12 Alkane (Emollient), Ascorbyl Glucoside (Skin brightening agent, Silica (Opacifying agent), Pyrus Malus (Apple) Fruit Extract (Skin conditioning agent), Hydrogenated Lecithin (Skin conditioning agent), Polyglyceryl-6 polyricinoleate (Emollient), Mannitol (Skin conditioning agent), Sodium Hydroxide (Solvent), Acrylates/C10-30 Alkyl Acrylate Crosspolymer (Viscosity controlling agent), Xanthan Gum (Emulsion stabilizer), Glutathione (Antioxidant), Ergothioneine (Antioxidant), Oryza Sativa (Rice) Bran Extract (Skin conditioning agent), Hydroethyl Acrylate/Sodium Acryloylmethyl Taurate Copolymer (Dispersing agent), Cetyl Alcohol (Emulsion stabilizer), Caprylyl Glycol (Skin conditioning agent), Lysine Carboxymethyl Cysteinate (Skin conditioning agent), Coco-Caprylate/Caprate (Skin conditioning agent), Tocopherol (Skin conditioning agent), Sodium Citrate (pH adjuster), Sodium Phytate (Chelating agent), Hexylene Glycol (Emulsifier), Ethylhexylglycerin (Skin conditioning agent), Helianthus Annuus (Sunflower) Extract (Emollient), Urea (Humectant), Caprylic/Capric Triglyceride (Skin conditioning agent), Sodium PCA (Skin conditioning agent), Rosmarinus Officinalis (Rosemary) Leaf Extract (Skin conditioning agent), Polysorbate 60 (Surfactant), Sorbitan Isostearate (Surfactant), Trehalose (Humectant), Ascorbic Acid (Skin conditioning agent), Glucose (Solvent), Vanilla Planifolia Fruit Extract (Skin conditioning agent), Polyquaternium-51 (Humectant), Triacetin (Solvent), Sodium Hyaluronate (Skin conditioning agent), Phenoxyethanol (Preservative)
[142] reported that IL-37 suppressed Th2 and Th17 responses but had no significant regulatory effects on Th1 or Treg responses
In vivo studies in EMs rodent models show that THP administration significantly reduces ectopic lesion volume and weight while also normalizing systemic inflammation by lowering serum TNF- levels and rebalancing epithelial-stromal crosstalk through modulation of epidermal growth factor (EGF) signaling, specifically by downregulating overexpressed EGF receptor (EGFR), phosphoinositide 3-kinase (PI3K), and serine/threonine kinase Akt (Wuputra et al., 2026)
What is vitamin B
