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Grx2 knockout or overexpression exacerbates or diminishes lipid peroxidation in human lens epithelial cells or opacity in mouse lenses, respectively, after treatment with ferroptosis inducers or UV-B via modulation of mitochondrial structure and the GSH/GSSG ratio [128]

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NLRP3 senses a wide array of endogenous and exogenous danger signals, including microbial products, environmental toxins, and host-derived DAMPs, making it a central player in inflammatory responses

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