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Kus-Likiewicz, M

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Because SELENOF has a thioredoxin-like catalytic site, SELENOF is thought to either regulate UGGT activity or the redox state of UGGT substrates (32)

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PMC 12313605

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Loss of progesterone responsiveness leads to the increased growth of endometriotic lesions and results in a non-receptive endometrium [31]

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Winger and Zamvil, 2016)

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