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The fact that this redox imbalance occurs as a consequence of GSH depletion may explain why several clinical trials have provided no evidence of significant cataract prevention arising from the administration of high dosages of ascorbic acid [13]

doi:10.1016/j.febslet.2014.05.046 Sitcheran R, Gupta P, Fisher PB, Baldwin AS (2005) Positive and negative regulation of EAAT2 by NF-kappaB: a role for N-myc in TNFalpha-controlled repression

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LTRPC2 Ca 2+ -permeable channel activated by changes in redox status confers susceptibility to cell death