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Nuclear localization of human SOD1 and mutant SOD1-specific disruption of survival motor neuron protein complex in transgenic amyotrophic lateral sclerosis mice

Similar content being viewed by others Introduction Glutathione (GSH) deficiency is one of the earliest biochemical perturbations in Parkinsons disease (PD), 1,2 leading to the hypothesis that GSH supplementation may have therapeutic value in alleviating PD symptoms or modifying progression

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Chronic stress does a number on the body and can lead to hormone imbalance, intestinal permeability (leaky gut), blood sugar imbalances, and a rapid depletion of glutathione reserves