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Researchers exploring metabolic disorders or muscle-wasting conditions often expose research models to IGF-1 LR3 as a model compound to investigate the role of IGF-1 signaling in nutrient utilization and energy homeostasis

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Similarly, ferroptosis inhibitors prevent high-glucose-induced cardiomyocyte dysfunction in diabetes-induced cardiomyopathy, suggesting that ferroptosis may play an essential part in the development of diabetic cardiomyopathy [12]

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