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GSH, the most abundant intracellular nonprotein thiol, plays a key role in intracellular defense, and decreased concentrations of this antioxidant are correlated with the increased frequency of reactive oxygen species (ROS)-mediated mitochondrial damage and apoptosis [9, 10]

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The same study described the mechanism as compound-specific , and the successful semaglutide system depends on close co-formulation, local concentration, tablet erosion, administration conditions, and a heavily characterized drug molecule