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in fact, these treatments promoted UPR mt and mitophagy, restoring mitochondrial and organismal function, and even stimulated the reduction of amyloid deposits in these models 114

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A tendon fibroblast study reported that BPC-157 increased growth hormone receptor expression in tendon fibroblasts at both mRNA and protein levels

it counteracted both early and delayed neurological damage (i.e., 24 and 72 hours after reperfusion)

Their effects on the individual components of MACE, myocardial infarction (reduced in LEADER and Harmony Outcomes), and stroke (reduced in SUSTAIN-6 and REWIND), varied in each trial (Table 1)