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When examined alongside BPC-157 and TB-500, researchers may explore how peptide signaling intersects with matrix remodeling, structural protein turnover, and tissue architecture maintenance under experimental conditions
Pharmacological inhibition of either Src or ERK for just 15 min resulted in rapid decline in -tubulin recruitment and microtubule nucleation when cells were adhered by wild-type integrins [17, 25], whereas molecular genetic approaches that resulted in the constitutive activation of Src or ERK restored the recruitment of -tubulin to the centrosome, as well as microtubule nucleation in cells adhered by mutant integrins [17, 25]
