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The previously mentioned study on rats with open ischemic wounds also reported an apparent decrease in wound area of the GHK-Cu group that was greater compared to the control group on days 3 to 5, 6 to 9, and 11 to 13

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8, 52, 53 Both mechanisms of action for BSO could be clinically important because previous studies have demonstrated that increased DNA crosslink/monoadducts and slow repair of DNA damage in L-PAM-treated patients is correlated to longer progression-free survival and improved outcome of treatment