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The fact that wild-type ATXN3 binds to components of the transcriptional machinery in cells, as well as the transcriptional changes reported in animal models of SCA3 in the presymptomatic phase, favors the theory of a general transcriptional dysregulation as an initiating factor of SCA3 [117,118,119]

Disclaimer: These statements are based on emerging research and are not intended to suggest diagnosis, treatment, or cure

Data Availability Statement The original contributions presented in the study are included in the article, further inquiries can be directed to the corresponding authors