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Thus, given the accumulating evidence across species implicating this pathway as integral to aging and its associated diseases, the development of therapeutics aimed at modulating IGF-1 signaling in humans could prove highly effective as a translational tool to delay aging

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For instance, differentiation of OL lineage cells generated from MS patient iPSCs occurs efficiently in vitro and in vivo 168 , yet is inhibited by exposure to peripheral blood mononuclear cell supernatants, with CD4+ T cell-derived interferon (IFN)- being a prime contributor 169

Inhibition of ACSL4 alleviates parkinsonism phenotypes by reduction of lipid reactive oxygen species

Maintaining optimal levels is essential for protecting cells from oxidative damage and supporting overall cellular resilience