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Peroxidase-like active nanomedicine with dual glutathione depletion property to restore oxaliplatin chemosensitivity and promote programmed cell death

This activation leads to the stimulation of the VEGFR2-Akt-eNOS signaling pathway, enhancing endothelial tube formation and blood flow recovery in ischemic tissues

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Blocking was then performed at room temperature for 30 min using serum blocking solution

In this review, we examine the structure and the stress regulation of the pathways involved in glutathione synthesis and degradation