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126: 1393407 Shear N, Spielberg S, Grant D, et al

These damages may include disruptions in spermatogenesis (e.g., genetic or developmental abnormalities) and testicular or post-testicular damage (e.g., gonadotoxins, hyperthermia, oxidants, and endocrine abnormalities)

Likewise, the counteraction of the isoprenaline myocardial infarction using BPC 157 therapy might include a NO effect [53]

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[3] Hypothalamic-Pituitary-Adrenal (HPA) Axis Modulation : DSIP has been observed to attenuate stress-induced corticotropin-releasing hormone (CRH) activity, potentially reducing systemic cortisol release