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The study further showed that restoring mitochondrial biogenesis through PGC-1 overexpression enhances T cell metabolic fitness and anti-tumor function, highlighting the critical role of metabolic regulation in determining T cell efficacy in the TME ( Moreover, hypoxic tumor cells reprogram their antioxidant systems to cope with elevated ROS levels

Studies using the chicken chorioallantoic membrane (CAM) assay and various rodent wound models have shown enhanced blood vessel formation in BPC-157-treated groups

L., Rodrigues, K

PPARgamma co-activator-1alpha (PGC-1alpha) reduces amyloid-beta generation through a PPARgamma-dependent