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ACSL4 is required to inhibit GPX4-induced ferroptosis, while ALOX12 is dispensable, whereas not required in p53-mediated ferroptosis, and therefore p53 promotes ferroptosis through a GPX4-independent pathway

doi: 10.1017/S0031182013000498 [DOI] [PubMed] [Google Scholar] 239

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At the onset of the protocol, the MMN of patients was significantly impaired compared to sex- and age- matched healthy controls ( p =0.003), without any evidence of concomitant P300 component deficits