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GSH biosynthesis is proposed to be regulated as a rheostat system that is designed to maintain sufficient antioxidant capacities, while preventing an overproduction of low molecular antioxidants that may interfere with the essential roles of ROS signaling (Shadel and Horvath, 2015)

Oxidative stress in polycystic ovarian syndrome and the effect of antioxidant N-acetylcysteine on ovulation and pregnancy rate

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Studies have reported that ATP produced through the mitochondrial oxidation of fatty acids from lipid droplets is an important source of energy for follicle and embryo development