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Elevated amount of ROS leads to prolonged oxidative stress and produces a potentially hazardous environment for the cells

Structural origin and rationale

Researchers should consult primary literature (Khavinson et al., 2003-2020) for protocol-specific concentrations

41589_2021_915_MOESM15_ESM.mpeg Supplementary Video 13 Representative movie for 100 ns MD simulation of oxidized GPX4 U46C (C46-SOOO-) with the camera zoomed in for a close-up of the active site

However, DHODHs role appears context dependent and debated, suggesting that patient stratification (for example, mitochondrial redox burden, GPX4/FSP1 status and metabolic state) will probably determine where DHODH inhibition provides ferroptosis-relevant benefit