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Using mouse Tc1, Tc17, and Tc22 T cells that had received in vitro differentiation, mass spectrometric metabolomic investigations were conducted to identify the metabolic causes of Tc22 polarization
Moreover, dysregulated lipid metabolism networks such as ACSL/SCD also drive EMT and migratory properties, and their pharmacologic inhibition selectively impairs CRC cell viability, further broadening the landscape of metabolic vulnerabilities in metastatic CRC [96]
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Moreover, MDA can react with amino acids and yield different adducts such as MDA-lysine [40], and MDA-glycine [70]
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