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Many preclinical studies have demonstrated that the expression of key enzymes for GSH synthesis (GCL, SLC7A11, GS) and GSSG reduction-related enzymes (G6PD, NADPH, and GR) is downregulated in myocardium exposed to pathological factors [40, 43,44,45,46,47,48,49,50]
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Antioxidant Mechanisms Laboratory studies indicate DSIP enhances cellular antioxidant capacity through several pathways that combat oxidative stress: Increased superoxide dismutase (SOD) activity Enhanced glutamate peroxidase function Improved mitochondrial respiratory activity Reduced stress protein synthesis Research shows DSIP pretreatment completely prevents hypoxia-induced reduction in mitochondrial function, suggesting powerful cellular protection mechanisms[1]
