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Permeabilization of the outer mitochondrial membrane via apoptosis regulator Bcl-2 associated X (BAX) and/or Bcl-2 homologous antagonist/killer (BAK), or the opening of the mitochondrial permeability transition pore (mPTP) in the inner mitochondrial membrane leading to the release of intrinsic apoptosis-induced factors, such as cytochrome c, is described to initiate the mitochondrial apoptotic pathway (93, 94)

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