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Underlying mechanisms may include chronic airway inflammation, impaired mucociliary clearance, increased bronchial hyperresponsiveness, increased development of T helper cell 2 (Th2) pathways relative to Th1 pathways, increased production of IgE, and greater allergic sensitization [9,25]

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CAFs were associated with a significant decrease in the expression of -smooth muscle actin, matrix metalloproteinase, and invasion abilities with WIN-55 treatment (Pietrovito et al