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Its primary mechanism of action involves increasing the expression of Brain-Derived Neurotrophic Factor (BDNF) in the cerebral cortex and hippocampus, promoting neuroplasticity, the formation of new synapses, and neuronal survival

Worth noting that this is a practice observation, not a controlled clinical trial

The 43-amino acid peptide demonstrates high binding affinity for G-actin monomers, preventing polymerisation through sequestration mechanisms

Sports 9(1):12 Funding Open access funding provided by Universit degli Studi di Torino within the CRUI-CARE Agreement

We tailor the formulation-topical or injectable-to your goals