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Agents that deplete bacterial GSH or inhibit its synthesis could sensitize bacteria to antibiotics, which makes resistant strains more treatable Exogenous GSH administration adds another dimension to therapeutic considerations

TNF-alpha-induced self-expression in human lung endothelial cells is inhibited by native and oxidized alpha1-antitrypsin

Pickart L, Thayer L, Thaler MM

For patients dealing primarily with low energy due to a B12 deficiency, standard B12 shots may be sufficient

Along this line of thought, it has been suggested that positively affecting the redox potential might benefit aspects neurologic functioning in the short term and may hold promise to contribute to attenuation of neurodegenerative processes that occur in MS over the longer term ( Glutathione is the principle naturally occurring antioxidant in neurons which protects against oxidative damage