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Chronic use leads to compensatory upregulation of NMDA and downregulation of GABA, causing CNS hyperexcitability on withdrawal the basis of alcohol withdrawal seizures and delirium tremens (F10.231, F10.232)

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However, the large proportion of CFTR expression deriving from other epithelial cell types and our observation of FOXI1 / CFTR decoupling by time course and knockout electrophysiology cautions against the simple model where cystic fibrosis is caused by loss of CFTR function in FOXI1 + ionocytes

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