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Based on in vitro findings, Hst1 decreased staining for senescence-associated -galactosidase activity and expression of mediators of senescence signaling, including p53, p21, and p16

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Across these models researchers have reported faster organisation of repair tissue, improved structural recovery, and enhanced fibroblast activity in injured animals, which together form the basis of the compounds widely studied regenerative profile

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All three routes improved MCL healing, supporting the clinical observation that BPC-157 retains activity orally and topically (a property unusual among peptides), although effect sizes varied by route