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An increase in temperature within PDAC tumor tissue drives alterations in lipid metabolism, including a decrease in PUFAs such as phosphatidylcholine plasmalogen (PC-P), attenuating lipid peroxidation and ferroptosis, and further drives gemcitabine resistance in PDAC through the p38-MAPK pathway [154]

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Studies document effects on 31.2% of human genes (approximately 4,000+ genes) with 50% expression change, suppressing RNA production in 70% of 54 genes overexpressed in metastatic colon cancer