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Additionally, inadequate performance of antioxidant systems can cause OS, which is linked to damage and oxidative modification of essential molecules, including proteins and lipids, ultimately damaging RBCs themselves or other vasculature components and, thus, impairing their functions (Figure 2) [55]

Small-molecule antagonists for CXCR2 and CXCR1 inhibit human melanoma growth by decreasing tumor cell proliferation, survival, and angiogenesis

Disinfecting and promoting the healing of wounds

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The oxidative stress-responsive muscle cells include: nuclear factor kappa B (NF-B), activator protein 1 (AP-1), Nrf2, and peroxisome proliferator-activated receptor gamma coactivator-1 alpha (PGC-1) [76]