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At the same time, it inhibits the PI3K/Akt/MAPK oncogenic pathway and suppresses CCNE1 transcriptional activation, thereby removing proliferative autonomy in ectopic stromal cells ( 2 (PGE 2 ) pathway, which is a central contributor to EMs-associated pain and disease progression, by upregulating 15-hydroxyprostaglandin dehydrogenase (HPGD), the rate-limiting enzyme responsible for catabolizing PGE 2 into inactive metabolites