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BAP1 reduces SLC7A11 expression by decreasing H2Aub occupancy on its promoter, leading to diminished cystine uptake, enhanced lipid peroxidation, and ferroptosis [109]

It upregulates heme oxygenase-1, reducing oxidative stress that impairs healing

This recently recognized mode of thiol-based regulation provides a detailed cysteine-based molecular mechanism and helps to explain how oxidative stress promotes inflammasome activity

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