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LTP induction also required activation of group I metabotropic glutamate receptors and PKC (Alle et al., 2001

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These prodrugs remain intact and inactive when circulating in plasma, that is, add profragments preferentially cut by enzymes enriched in the tumor (such as histone deacetylase (HDAC) or cathepsin), and then selectively release DON in the target tissue, thereby reducing the dose and increasing the therapeutic index

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