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Also, a different rat model of a low protein diet reported upregulation of genes involved in de novo lipogenesis (SREBP1C, FASN, ACC, and SCD1), and downregulation of genes associated with mitochondrial dysfunction (PGC1, TIM23, AND TFAM), contributing to liver steatosis and fibrosis [136]

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Together, they address both structural (fibrosis, neuronal damage) and functional (inflammation, oxidative stress) aspects of disease

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