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By comparison, in the BM microenvironment affected by peripheral inflammation, N1 polarization is orchestrated by systemic IFN-I signaling and is precisely regulated through a newly identified IFN-IChac2GSH signaling axis

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The abundance of almost all immune cells was negatively correlated with the expression of marker genes of acini (mainly serous acini) and progenitor cells and the mitochondrial biogenesis-related gene PPARGC1A