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It has been proposed that depletion of hepatic GSH, particularly mitochondrial GSH, is one of the early changes associated with chronic alcohol consumption and is a critical contributor to ALD pathogenesis 11

Moreover, the LA strain elevates liposome content and COX-2 expression, which is associated with TLR2 and TLR6 expression, respectively (141, 142)

Cell 185 (14), 24012421

In addition, there was a body of evidence implicating nonmitochondrial enzymes such as NADPH oxidases, nitric oxide (NO) synthases and heme oxygenases located in glomus cells [79]