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Chenodeoxycholic acid stimulates Cl(-) secretion via cAMP signaling and increases cystic fibrosis transmembrane conductance regulator phosphorylation in T84 cells

This understanding shifts therapy from late-stage symptom care to early root cause correction

Studies examine: VEGFR2 activation pathways and intracellular adaptor protein recruitment Focal adhesion kinase (FAK) phosphorylation mechanisms Paxillin recruitment and integrin-actin cytoskeleton interactions Endothelial cell proliferation and migration assays in vitro Research models study the combined effects of VEGFR2-mediated proliferation and FAK-paxillin-mediated cellular motility in angiogenesis pathway investigations

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